In this study, we aimed to estimate the relationship between some common bean traits using molecular markers and applying QTL mapping. We used a segregating population derived from a crossing between common bean cultivars, Jalo and Small White, in the Southern State of Minas Gerais. Of F2 plants, 190 F2:3 progenies were generated. Phenotypic measures related to the .. Read More»
Genet. Mol. Res. 16(3): gmr16039686
DOI: 10.4238/gmr16039686
The Orchidaceae represent one of the largest and most diverse families on the planet. However, this family is constantly threatened by predators and by the advancement of urban centers over its natural habitats. The objective of this study was to use inter-simple sequence repeat markers to evaluate the genetic diversity between orchid accessions of the Laeliinae sub.. Read More»
Genet. Mol. Res. 15(2): gmr.15027997
DOI: 10.4238/gmr.15027997
We aimed to identify simple sequence repeat (SSR) markers linked to quantitative trait loci (QTLs) associated with white mold resistance in a segregating population derived from a cross between common bean cultivars Jalo and Small White, in the Southern State of Minas Gerais. Parents were crossed to obtain the F2 generation of 190 plants. From these, F2:3 and F2:4 progenies were obtained for ph.. Read More»
Genet. Mol. Res. 15(3): gmr.15038724
DOI: 10.4238/gmr.15038724
The use of genetically resistant cultivars is the best method to reduce losses caused by white mold (Sclerotinia sclerotiorum (Lib.) de Bary). As the best known resistance sources are not adapted, the genetic control of white mold in the common bean must be understood to guide breeding more efficiently. The objective of the present study was to identify the genetic .. Read More»
Genet. Mol. Res. 7(3): vol7-3gmr466
DOI: 10.4238/vol7-3gmr466
The interaction between polygalacturonase-inhibiting proteins (PGIPs), produced by plants, and endopolygalacturonases (PGs), produced by fungi, limits the destructive potential of PGs and can trigger plant defense responses. This study aimed to i) investigate variation in the expression of different common bean (Phaseolus vulgaris L.) genotypes and its relationship with resistance to white mold.. Read More»
Genet. Mol. Res. 15(3): gmr.15038269
DOI: 10.4238/gmr.15038269
In many species, low levels of polymorphism prevent the assembly of linkage maps that are used to identify genetic markers related to the expression of quantitative trait loci (QTLs). This study compared two methods of locating QTLs in association studies that do not require a previous estimation of linkage maps. Method I (MI) was a Bayesian multiple marker regression and Method II (MII) combin.. Read More»
Genet. Mol. Res. 14(3): http://dx.doi.org/2015.September.25.13
DOI: http://dx.doi.org/10.4238/2015.September.25.13
In this study, we identified simple sequence repeat, amplified fragment length polymorphism, and sequence-related amplified polymorphism markers linked to quantitative trait loci (QTLs) for resistance to white mold disease in common bean progenies derived from a cross between lines CNFC 9506 and RP-2, evaluated using the oxalic acid test and using Bayesian analysis. DNA was extracted .. Read More»
Genet. Mol. Res. 14(1): 2015.February.6.16
DOI: 10.4238/2015.February.6.16
We examined the capacity of strains of Glomerella cin-gulata f. sp phaseoli fungus (Colletotrichum lindemuthianum sexual stage) to form recombinants, using random amplified polymorphic DNA (RAPD). Crosses of all possible combinations between strains 40, 42, 20, 21, 22, 23, 24, 25, and 26 were made on Petri dishes using M3 culture medium. The 42 x 21 cross produced t.. Read More»
Genet. Mol. Res. 6(3):
The aims of this study were to evaluate the reaction of common bean lines to white mold, the aggressiveness of different Sclerotinia sclerotiorum isolates from various common bean production areas in Brazil, and comparison of the diallel and GGE (genotype main effect plus genotype-by-environment interaction) biplot analysis procedures via study of the line-by-isolate interaction. Eleven common .. Read More»
Genet. Mol. Res. 13(4):
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