Staphylococcus Aureus
Differential expression of genes associated with low-level vancomycin resistance in Staphylococcus aureus
Authors: Auttawit Sirichoat, Viraphong Lulitanond, Sujintana Wongthong, Aroonwadee Chanawong, Ratree Tavichakorntrakool and Aroonlug Lulitanond
Heterogeneous vancomycin-intermediate Staphylococcus aureus (hVISA) and vancomycin-intermediate S. aureus (VISA) have been associated with treatment failure, and hVISA cannot be detected by routine disc diffusion test. Currently, there is no specific genetic marker for detection of these strains. The aim of the current study was to inves.. Read More»
- Genet. Mol. Res. 18(2):
Phenotypic and genotypic characterization of Staphylococcus aureus isolates in milk from flocks diagnosed with subclinical mastitis
Authors: A.R.E.O. Xavier, A.C. Almeida, C.N. Souza, L.M.V Silva, A.X.A. Ruas, D.A. Sanglard, A.F.M. J�ºnior , A.M.E. Oliveira and M.A.S. Xavier
The Staphylococcus aureus is the most common isolated microorganism in ruminant animal species diagnostic with clinical or subclinical mastitis. Dairy herds with these diseases can transfer S. aureus into the milk supply, which can lead to food poisoning in humans. The objective of this study was to evaluate the profile of ant.. Read More»
- Genet. Mol. Res. 16(2):
- gmr16029709
- DOI:
- 10.4238/gmr16029709
Methylation analysis of CXCR1 in mammary gland tissue of cows with mastitis induced by Staphylococcus aureus
Authors: Y.J. Mao, X.R. Zhu, R. Li, D. Chen, S.Y. Xin, Y.H. Zhu, X.X. Liao, X.L. Wang, H.M. Zhang, Z.P. Yang and L.G. Yang
Mastitis is the most important disease in the global dairy industry, and causes large economic losses. Staphylococcus aureus is one of most common pathogens that cause bovine mastitis. CXCR1 has been implicated as a prospective genetic marker for mastitis resistance in dairy cows; CXCR1 expression significantly increases when .. Read More»
- Genet. Mol. Res. 14(4):
- 2015.October.19.4
- DOI:
- 10.4238/2015.October.19.4
Molecular analysis and frequency of Staphylococcus aureus virulence genes isolated from bloodstream infections in a teaching hospital in Tianjin, China
Authors: L.X. Wang Z.D. Hu Y.M. Hu B. Tian Jing Li F.X. Wang H. Yang H.R. Xu Y.C. Li J. Li
Staphylococcus aureus is an important cause of bloodstream infections worldwide. We examined the prevalence of genes that encode erythromycin ribosome methylase and bacterial toxins in S. aureus collected from bloodstream infections. Sixty different S. aureus isolates were obtained from blood cultures of patients who were admitted to a Teaching Hospital i.. Read More»
- Genet. Mol. Res. 12(1):
- 2013.March.11.12
- DOI:
- 10.4238/2013.March.11.12
Evaluation of methods of DNA extraction from Staphylococcus aureus in milk for use in real-time PCR
Authors: A.G. Dibbern, B.G. Botaro, M.P. Viziack, L.F.P. Silva and M.V. Santos
The aim of this study was to evaluate the repeatability and performance of 4 methods of extracting DNA from Staphylococcus aureus (SAU) and the gene encoding bovine mitochondrial cytochrome B (BMCB) in milk samples from cows with subclinical mastitis for use in amplification by real-time polymerase chain reaction. Two milk samples were obtained from cows .. Read More»
- Genet. Mol. Res. 14(1):
- 2015.January.16.6
- DOI:
- 10.4238/2015.January.16.6
An experimental model for Staphylococcus aureus hepatic abscess in Bama minipig
Authors: R.G. Zhang, X.D. Wang*, X.L. Zhang and Y.S. Yang
Pyogenic hepatic abscess (PHA) is a rare but potentially serious disease. Investigations of new therapeutic methods urgently need experimental support in corresponding animal models. However, to date, few studies have evaluated PHA in the minipig. The linear regression equation of the Staphylococcus aureus ATCC 25923 .. Read More»
- Genet. Mol. Res. 13(3):
- 2014.February.21.12
- DOI:
- 10.4238/2014.February.21.12
Distribution of food-borne Staphylococcus aureus enterotoxin genes
Authors: W.D. Hu
We identified and analyzed 5 new-type enterotoxin genes, including SEj, SEl, SEq, SEm, and SEr, to explore the distribution of 5 enterotoxin genes in Staphylococcus aureus of different origins as well as their correlations and differences. We examined the distribution of the S. aureus enterotoxin genes and their pathogenic mechanisms. A total of 660 speci.. Read More»
- Genet. Mol. Res. 15(1):
- gmr.15016084
- DOI:
- 10.4238/gmr.15016084
Evaluation of the humoral immune response in BALB/c mice immunized with a naked DNA vaccine anti-methicillin-resistant Staphylococcus aureus
Authors: D.M. Roth, J.P.M. Senna and D.C. Machado
Methicillin-resistant Staphylococcus aureus (MRSA) is the major pathogen involved in nosocomial infections, leading to high rates of morbidity and mortality in hospitals worldwide. The methicillin resistance occurs due to the presence of an additional penicillin-binding protein, PBP2a, which has low affinity for β-lactam .. Read More»
- Genet. Mol. Res. 5(3):
Epidemiological investigation of community-acquired Staphylococcus aureus infection
Authors: W. Jiang, Z. Zhou, K. Zhang and Y. Yu
The current study aimed to reveal the pathogenic and spreading mechanisms of community-acquired Staphylococcus aureus (CA-SA) by analyzing its prevalence, drug resistance, virulence, and pathogenic factors. Historical information regarding CA skin and soft tissue infections were collected and disease characteristics were analyzed. Isolated CA-SA strains w.. Read More»
- Genet. Mol. Res. 12(4):
- 2013
- DOI:
- 10.4238/2013
Rifampicin resistance among multi-resistant MRSA clinical isolates from Chennai, India, and their molecular characterization
Authors: K. Murugan, K. Kavitha and S. Al-Sohaibani
High-level methicillin-resistant Staphylococcus aureus (MRSA) isolates show rapid evolution of rifampicin resistance, forcing reliance upon expensive and often inferior antibiotics to manage these infections. Accordingly, this study was conducted to: 1) evaluate the level of multidrug resistance among hospital-associated MRSA isolates from Chennai, India;.. Read More»
- Genet. Mol. Res. 14(1):
- http://dx.doi.org/2015.March.31.1
- DOI:
- http://dx.doi.org/10.4238/2015.March.31.1